Tumor necrosis factor-α produced in cardiomyocytes mediates a predominant myocardial inflammatory response to stretch in early volume overload-Reference-Cited by-同舟云学术

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Tumor necrosis factor-α produced in cardiomyocytes mediates a predominant myocardial inflammatory response to stretch in early volume overload

Published:2010-07 Issue:1 Volume:49 Page:70-78
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Chen Yuanwen,Pat Betty,Zheng Junying,Cain Laura,Powell Pamela,Shi Ke,Sabri Abdelkarim,Husain Ahsan,Dell'Italia Louis J.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference32 articles.

1. Pharmacologic inhibition of mast cell degranulation prevents left ventricular remodeling induced by chronic volume overload in rats;Brower;J. Card. Fail.,2005

2. Left ventricular eccentric remodeling and matrix loss are mediated by bradykinin and precede cardiomyocyte elongation in rats with volume overload;Ryan;J. Am. Coll. Cardiol.,2007

3. Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient rats;Levick;J. Mol. Cell. Cardiol.,2008

4. Cause and effect relationship between myocardial mast cell number and matrix metalloproteinase activity;Brower;Am. J. Physiol. Heart Circ. Physiol.,2002

5. Cardiac-specific overexpression of tumor necrosis factor-alpha causes lethal myocarditis in transgenic mice;Kubota;J. Card. Fail.,1997

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