A hotspot in the glucocorticoid receptor DNA-binding domain susceptible to loss of function mutation
Author:
Funder
NIH
Publisher
Elsevier BV
Subject
Organic Chemistry,Clinical Biochemistry,Pharmacology,Endocrinology,Molecular Biology,Biochemistry
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4. Glucocorticoid receptor heterozygosity combined with lack of receptor auto-induction causes glucocorticoid resistance in Jurkat acute lymphoblastic leukemia cells;Riml;Cell Death Differ,2004
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