C-type natriuretic peptide does not attenuate the development of pulmonary hypertension caused by hypoxia and VEGF receptor blockade-Reference-Cited by-同舟云学术

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C-type natriuretic peptide does not attenuate the development of pulmonary hypertension caused by hypoxia and VEGF receptor blockade

Published:2011-09 Issue:13-14 Volume:89 Page:460-466
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Casserly Brian,Mazer Jeffrey M.,Vang Alexander,Harrington Elizabeth O.,Klinger James R.,Rounds Sharon,Choudhary Gaurav

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference45 articles.

1. Formation of plexiform lesions in experimental severe pulmonary arterial hypertension;Abe;Circulation,2010

2. Marrow cell infusion attenuates vascular remodeling in a murine model of monocrotaline-induced pulmonary hypertension;Aliotta;Stem Cells Dev,2009

3. Mechanisms of action of atrial natriuretic factor and C-type natriuretic peptide;Amin;Hypertension,1996

4. Natriuretic peptide receptor-C signaling and regulation;Anand-Srivastava;Peptides,2005

5. Endothelial dysfunction in pulmonary hypertension;Budhiraja;Circulation,2004

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1. CNP, the Third Natriuretic Peptide: Its Biology and Significance to the Cardiovascular System;Biology;2022-06-29

2. α7 Nicotinic acetylcholine receptor mediates right ventricular fibrosis and diastolic dysfunction in pulmonary hypertension;JCI Insight;2021-06-22

3. C-Type Natriuretic Peptide Ameliorates Lipopolysaccharide-Induced Cardiac Dysfunction in Rats with Pulmonary Arterial Hypertension;BioMed Research International;2018-12-20

4. New dynamic viewing of mast cells in pulmonary arterial hypertension (PAH): contributors or outsiders to cardiovascular remodeling;Journal of Thoracic Disease;2018-05

5. Effect of α7 nicotinic acetylcholine receptor activation on cardiac fibroblasts: a mechanism underlying RV fibrosis associated with cigarette smoke exposure;American Journal of Physiology-Lung Cellular and Molecular Physiology;2017-05-01

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