Menadione-induced oxidative stress in hepatocytes isolated from fed and fasted rats: The role of NADPH-regenerating pathways

Author:

Smith Peter F.,Alberts David W.,Rush Glenn F.

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference47 articles.

1. Protective role of the glutathione redox cycle against adriamycin-mediated toxicity in isolated hepatocytes;Babson;Biochem. Pharmacol,1981

2. A general mechanism for microsomal activation of quinone anticancer agents to free radicals;Bachur;Cancer Res,1978

3. NADPH cytochrome P-450 reductase activation of quinone anticancer agents to free radicals;Bachur,1979

4. Rates of pentose cycle flux in perfused rat liver: Evolution of the role of reducing equivalents from the pentose cycle for mixed-function oxidation;Belinsky;Mol. Pharmacol,1985

5. The metabolism of menadione impairs the ability of rat liver mitochondria to take up and retain calcium;Bellomo;J. Biol. Chem,1982

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