The role of Na+,K+-ATPase on TBI-induced physiopathology-Reference-Cited by-同舟云学术

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The role of Na+,K+-ATPase on TBI-induced physiopathology

Published:2022 Issue: Volume: Page:195-205
ISSN:
Container-title:Cellular, Molecular, Physiological, and Behavioral Aspects of Traumatic Brain Injury
language:
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Author:

Royes Luiz Fernando Freire,Fighera Michele Rechia,Furian Ana Flavia,Oliveira M.S.,Rambo Leonardo Magno,Nascimento Alexandre Seixas

Publisher

Elsevier

Reference63 articles.

1. Anatomical and physiological evidence for D1 and D2 dopamine receptor colocalization in neostriatal neurons;Aizman;Nature Neuroscience,2000

2. Functional interaction between Na/K-ATPase and NMDA receptor in cerebellar neurons;Akkuratov;Molecular Neurobiology,2015

3. A specific and essential role for Na,K-ATPase α3 in neurons co-expressing α1 and α3;Azarias;Journal of Biological Chemistry,2013

4. Cellular alterations in human traumatic brain injury: Changes in mitochondrial morphology reflect regional levels of injury severity;Balan;Journal of Neurotrauma,2013

5. FXYD7 is a brain-specific regulator of Na, K-ATPase α1-β isozymes;Béguin;EMBO Journal,2002

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