‘Late’ multiorgan failure in major burns: A “three-event” construct rather than a “two-event” construct
Author:
Publisher
Elsevier BV
Subject
Critical Care and Intensive Care Medicine,Emergency Medicine,General Medicine,Surgery
Reference31 articles.
1. The immunological consequences of injury;Ni-choileain;Surgeon,2006
2. Multiorgan failure. Pathophysiology and potential future therapy;Deitch;Ann Surg,1992
3. The two-event construct of post injury multiple organ failure;Moore;Shock,2005
4. Burns, bacterial translocation, gut barier function and failure;Magnetti;J Burn Care Rabil,2005
5. Burn injury exacerbates hemodynamic and metabolic responses in rats with polymicrobial sepsis;Goto;J Burn Care Res,2006
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. The Role of Th-17 Cells and γδ T-Cells in Modulating the Systemic Inflammatory Response to Severe Burn Injury;International Journal of Molecular Sciences;2017-04-03
2. Th17 (IFNγ- IL17+) CD4+ T Cells Generated After Burn Injury May Be a Novel Cellular Mechanism for Postburn Immunosuppression;Journal of Trauma: Injury, Infection & Critical Care;2011-03
3. Extracorporeal shock wave therapy suppresses the early proinflammatory immune response to a severe cutaneous burn injury;International Wound Journal;2009-02
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