Diabetic cataracts-Reference-Cited by-同舟云学术

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Diabetic cataracts

Published:2021-05 Issue:5 Volume:67 Page:101134
ISSN:0011-5029
Container-title:Disease-a-Month
language:en
Short-container-title:Disease-a-Month

Author:

Greenberg Mitchell J.,Bamba Sonya

Publisher

Elsevier BV

Subject

General Medicine

Reference6 articles.

1. https://pdfs.semanticscholar.org/dd1d/7dd7992ae67546b22744f1feaf5d63aa2b95.pdf

2. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6173035/

3. https://www.wjgnet.com/1948-9358/full/v10/i3/140.htm

4. https://www.hindawi.com/journals/joph/2010/608751/

5. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3589218/

Cited by 11 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. RBM15 Promotes High Glucose-Induced Lens Epithelial Cell Injury by Inducing PRNP N6-Methyladenine Modification During Diabetic Cataract;Current Eye Research;2024-08-29

2. Mitofusin 2 inhibits high glucose-induced apoptosis of human lens epithelial cells via modulating mitochondrial function and autophagy;Folia Histochemica et Cytobiologica;2024-07-30

3. SP1 promotes high glucose-induced lens epithelial cell viability, migration and epithelial-mesenchymal transition via regulating FGF7 and PI3K/AKT pathway;International Ophthalmology;2024-07-06

4. Melatonin, ATP, and Cataracts: The Two Faces of Crystallin Phase Separation;2024-03-17

5. Methyltransferase METTL3‐mediated maturation of miR‐4654 facilitates high glucose‐induced apoptosis and oxidative stress in lens epithelial cells via decreasing SOD2;Chemical Biology & Drug Design;2024-02

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