ZNF750 Is a p63 Target Gene that Induces KLF4 to Drive Terminal Epidermal Differentiation-Reference-Cited by-同舟云学术

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ZNF750 Is a p63 Target Gene that Induces KLF4 to Drive Terminal Epidermal Differentiation

Published:2012-03 Issue:3 Volume:22 Page:669-677
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Sen George L.,Boxer Lisa D.,Webster Dan E.,Bussat Rose T.,Qu Kun,Zarnegar Brian J.,Johnston Danielle,Siprashvili Zurab,Khavari Paul A.

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference22 articles.

1. Seborrhea-like dermatitis with psoriasiform elements caused by a mutation in ZNF750, encoding a putative C2H2 zinc finger protein;Birnbaum;Nat. Genet.,2006

2. p63 in epithelial survival, germ cell surveillance, and neoplasia;Crum;Annu. Rev. Pathol.,2010

3. Deletion of the late cornified envelope LCE3B and LCE3C genes as a susceptibility factor for psoriasis;de Cid;Nat. Genet.,2009

4. Perp is a p63-regulated gene essential for epithelial integrity;Ihrie;Cell,2005

5. p63 in skin development and ectodermal dysplasias;Koster;J. Invest. Dermatol.,2010

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2. ABCC1 Is a ΔNp63 Target Gene Overexpressed in Squamous Cell Carcinoma;International Journal of Molecular Sciences;2024-08-10

3. Skin Development and Disease: A Molecular Perspective;Current Issues in Molecular Biology;2024-07-30

4. A loss-of-function variant in KLF4 affecting zinc finger motifs causes progressive symmetric erythrokeratodermia;British Journal of Dermatology;2024-06-28

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