Deletion of adenosine A1 or A2A receptors reduces l-3,4-dihydroxyphenylalanine-induced dyskinesia in a model of Parkinson's disease-Reference-Cited by-同舟云学术

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Deletion of adenosine A1 or A2A receptors reduces l-3,4-dihydroxyphenylalanine-induced dyskinesia in a model of Parkinson's disease

Published:2011-01 Issue: Volume:1367 Page:310-318
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Xiao Danqing,Cassin Jared J.,Healy Brian,Burdett Thomas C.,Chen Jiang-Fan,Fredholm Bertil B.,Schwarzschild Michael A.

Funder

NIH

DoD

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference53 articles.

1. Modulation of Ca2+ channels by activation of adenosine A1 receptors in rat striatal glutamatergic nerve terminals;Ambrosio;Neurosci. Lett.,1996

2. Mutant mice and neuroscience: recommendations concerning genetic background. Banbury Conference on genetic background in mice;Banbury Conference;Neuron,1997

3. A crucial role for forebrain adenosine A(2A) receptors in amphetamine sensitization;Bastia;Neuropsychopharmacology,2005

4. Pathophysiology of l-dopa-induced dyskinesias;Bedard;Mov. Disord.,1999

5. Neurotransmitter receptor analysis in transgenic mouse models;Benn;Meth. Mol. Biol.,2004

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