Protective effect of scallop-derived plasmalogen against vascular dysfunction, via the pSTAT3/PIM1/NFATc1 axis, in a novel mouse model of Alzheimer’s disease with cerebral hypoperfusion-Reference-Cited by-同舟云学术

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Protective effect of scallop-derived plasmalogen against vascular dysfunction, via the pSTAT3/PIM1/NFATc1 axis, in a novel mouse model of Alzheimer’s disease with cerebral hypoperfusion

Published:2024-04 Issue: Volume:1828 Page:148790
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Zhai Yun^ORCID,Morihara Ryuta,Feng Tian,Hu Xinran,Fukui Yusuke,Bian Zhihong,Bian Yuting,Yu Haibo,Sun Hongming,Takemoto Mami,Nakano Yumiko,Yunoki Taijun,Tang Ying,Ishiura Hiroyuki,Yamashita Toru

Funder

Japan Agency for Medical Research and Development

Harbin Medical University

Publisher

Elsevier BV

Reference58 articles.

1. Aguilar-Pineda, J.A., Vera-Lopez, K.J., Shrivastava, P., Chávez-Fumagalli, M.A., Nieto-Montesinos, R., Alvarez-Fernandez, K.L., Goyzueta Mamani, L.D., Davila Del-Carpio, G., Gomez-Valdez, B., Miller, C.L., Malhotra, R., Lindsay, M.E., Lino Cardenas, C.L., 2021. Vascular smooth muscle cell dysfunction contribute to neuroinflammation and Tau hyperphosphorylation in Alzheimer disease. iScience. 24, 102993.

2. Hypoperfusion, Mitochondria Failure, Oxidative Stress, and Alzheimer Disease;Aliev;J. Biomed. Biotechnol.,2003

3. The JAK/STAT3 pathway is a common inducer of astrocyte reactivity in Alzheimer's and Huntington's diseases;Ben Haim;J. Neurosci.,2015

4. Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid;Calhoun;PNAS,1999

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