Arc regulates brain damage and neuroinflammation via Sirt1 signaling following subarachnoid hemorrhage
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference41 articles.
1. Immediate-early genes modulation by antipsychotics: translational implications for a putative gateway to drug-induced long-term brain changes;de Bartolomeis;Front Behav. Neurosci.,2017
2. The immediate early gene arc/arg3.1: regulation, mechanisms, and function;Bramham;J. Neurosci.,2008
3. The AMPAR antagonist perampanel attenuates traumatic brain injury through anti-oxidative and anti-inflammatory activity;Chen;Cell Mol. Neurobiol.,2017
4. Glutamate-induced rapid induction of Arc/Arg3.1 requires NMDA receptor-mediated phosphorylation of ERK and CREB;Chen;Neurosci. Lett.,2017
5. RNF216 mediates neuronal injury following experimental subarachnoid hemorrhage through the Arc/Arg3.1-AMPAR pathway;Chen;FASEB J.,2020
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4. NAD-Dependent Protein Deacetylase Sirtuin-1 Mediated Mitophagy Regulates Early Brain Injury After Subarachnoid Hemorrhage;Journal of Inflammation Research;2024-03
5. Injection of Collagen Binding Domain-Brain Derived Neurotrophic Factor Promotes SIRT1 Expression: Improving Neuroinflammation in Experimental Subarachnoid Hemorrhage;Discovery Medicine;2024
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