Repeated psychosocial stress causes glutamic acid decarboxylase isoform-67, oxidative-Nox-2 changes and neuroinflammation in mice: Prevention by treatment with a neuroactive flavonoid, morin-Reference-Cited by-同舟云学术

Repeated psychosocial stress causes glutamic acid decarboxylase isoform-67, oxidative-Nox-2 changes and neuroinflammation in mice: Prevention by treatment with a neuroactive flavonoid, morin

Published:2020-10 Issue: Volume:1744 Page:146917
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Ben-Azu Benneth^ORCID,Emokpae Osagie,Ajayi Abayomi Mayowa,Jarikre Thiophilus Aghogho,Orhode Valiant,Aderibigbe Adegbuyi Oladele,Umukoro Solomon,Iwalewa Ezekiel O.

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference77 articles.

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2. Interleukin-6 mediates the increase in NADPH oxidase in the ketamine model of schizophrenia;Behrens;J. Neurosci.,2008

3. Morin pretreatment attenuates schizophrenia-like behaviors in experimental animal models;Ben-Azu;Drug Res.,2017

4. Probable mechanisms involved in the antipsychotic-like activity of morin in mice;Ben-Azu;Biomed. Pharmacother.,2018

5. Morin attenuates neurochemical changes and increased oxidative/nitrergic stress in brains of mice exposed to ketamine: prevention and reversal of schizophrenia-like symptoms;Ben-Azu;Neurochem. Res.,2018

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