Diannexin alleviates myocardial ischemia–reperfusion injury by orchestrating cardiomyocyte oxidative damage, macrophage polarization and fibrotic process by TLR4-NF-kB-mediated inactivation of NLRP3 inflammasome-Reference-Cited by-同舟云学术

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Diannexin alleviates myocardial ischemia–reperfusion injury by orchestrating cardiomyocyte oxidative damage, macrophage polarization and fibrotic process by TLR4-NF-kB-mediated inactivation of NLRP3 inflammasome

Published:2024-03 Issue: Volume:130 Page:111668
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Zhang Lin^ORCID,Zhao Songlin,Wang Yaqi

Publisher

Elsevier BV

Reference37 articles.

1. l-Carnitine and heart disease;Wang;Life Sci.,2018

2. Evolving therapies for myocardial ischemia/reperfusion injury;Ibanez;J. Am. Coll. Cardiol.,2015

3. Pathophysiology of myocardial infarction and acute management strategies;Tibaut;Cardiovasc. Hematol. Agents Med. Chem.,2017

4. Myocardial reperfusion injury;Yellon;N. Engl. J. Med.,2007

5. Role of oxidative stress in reperfusion following myocardial ischemia and its treatments;Xiang;Oxid. Med. Cell. Longev.,2021

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. KLF4‐induced upregulation of SOCS1 ameliorates myocardial ischemia/reperfusion injury by attenuating AC16 cardiomyocyte damage and enhancing M2 macrophage polarization;Journal of Biochemical and Molecular Toxicology;2024-08-26

2. Levosimendan and Dobutamin Attenuate LPS-Induced Inflammation in Microglia by Inhibiting the NF-κB Pathway and NLRP3 Inflammasome Activation via Nrf2/HO-1 Signalling;Biomedicines;2024-05-03

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