Transcription factor ZBP-89 in cancer growth and apoptosis
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Genetics,Oncology
Reference100 articles.
1. ZBP-89-induced apoptosis is p53-independent and requires JNK;Bai;Cell Death Differ.,2004
2. ZBP-89 promotes growth arrest through stabilization of p53;Bai;Mol. Cell. Biol.,2001
3. Transcription factor ZBP-89 cooperates with histone acetyltransferase p300 during butyrate activation of p21waf1 transcription in human cells;Bai;J. Biol. Chem.,2000
4. ATM phosphorylates ZBP-89 at Ser202 to potentiate p21waf1 induction by butyrate;Bai;Biochem. Biophys. Res. Commun.,2007
5. Three zinc finger nuclear proteins, Sp1, Sp3, and a ZBP-89 homologue, bind to the cyclic adenosine monophosphate-responsive sequence of the bovine adrenodoxin gene and regulate transcription;Cheng;Biochemistry,2000
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