Transition of cervical carcinoma in situ to invasive cancer: Role of p16INK4a expression in progression and in recurrence

Author:

Anschau Fernando,Schmitt Virgínia Minghelli,Lambert Ana Paula Franco,Gonçalves Manoel Afonso Guimarães,Machado Denise Cantarelli

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Molecular Biology,Pathology and Forensic Medicine

Reference32 articles.

1. Persistent human papillomavirus infection and smoking increased risk of failure of treatment of cervical intraepithelial neoplasia (CIN);Alcadious;Int. J. Cancer,2002

2. Codon 72 polymorphism of the TP53 gene;Ara;Nucleic Acid Res.,1990

3. FIGO staging classifications and clinical practice guidelines in the management of gynecologic cancers;Benedet;FIGO Committee on Gynecologic Oncology. Int. J. Gynaecol. Obstet.,2000

4. Expression of p16(INK4a) and cervical infection with high-risk human papillomavirus are not related to p53 activity in cervical intraepithelial neoplasia;Bragança;Int. J. Gynecol. Cancer,2007

5. P16INK4a expression is related to grade of CIN and high-risk human papillomavirus but does not predict virus clearance after conization or disease outcome;Branca;Int. J. Gynecol. Pathol.,2004

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