Redox regulation by NRF2 in aging and disease-Reference-Cited by-同舟云学术

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Redox regulation by NRF2 in aging and disease

Published:2019-04 Issue: Volume:134 Page:702-707
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Schmidlin Cody J.,Dodson Matthew B.,Madhavan Lalitha,Zhang Donna D.

Funder

National Institute of Environmental Health Sciences

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference50 articles.

1. ROS, cell senescence, and novel molecular mechanisms in aging and age-related diseases;Davalli;Oxidative Med. Cell. Longev.,2016

2. ROS function in redox signaling and oxidative stress;Schieber;Curr. Biol.,2014

3. The Nrf2 regulatory network provides an interface between redox and intermediary metabolism;Hayes;Trends Biochem. Sci.,2014

4. Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain;Itoh;Genes Dev.,1999

5. p97 negatively regulates NRF2 by extracting ubiquitylated NRF2 from the KEAP1-CUL3 E3 complex;Tao;Mol. Cell. Biol.,2017

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