Calcitriol attenuates the basal and vasoactive intestinal peptide-stimulated cAMP production in prolactin-secreting rat pituitary (GH4C1) cells
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Molecular Biology,Biochemistry
Reference34 articles.
1. Stoichiometry of receptor‐G s ‐adenylate cyclase interactions
2. Cholecalciferol metabolites attenuate cAMP production in rat thyroid cells (FRTL-5)
3. The mechanisms by which vasoactive intestinal peptide (VIP) and thyrotropin releasing hormone (TRH) stimulate prolactin release from pituitary cells
4. Vasoactive intestinal peptide and peptide with N-terminal histidine and C-terminal isoleucine increase prolactin secretion in cultured rat pituitary cells (GE4C1) via a cAMP-dependent mechanism which involves transient elevation of intracellular Ca2+
5. Binding and degradation of vasoactive intestinal peptide in prolactin-producing cultured rat pituitary tumour cells (GH4C1)
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Vitamin D: A Hormonal Regulator of the cAMP Signaling Pathway;Critical Reviews in Biochemistry and Molecular Biology;1999-01
2. Modulatory role of 1,25 dihydroxyvitamin D3on pancreatic islet insulin release via the cyclic AMP pathway in the rat;British Journal of Pharmacology;1997-06
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