Detection of Human Papillomavirus 16 Transcription in Human Prostate Tissue
Author:
Affiliation:
1. Manitoba Institute of Cell Biology, Departments of Physiology, Pathology and Medical Microbiology, University of Manitoba and Cadham Provincial Laboratory, Winnipeg, Manitoba
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Urology
Reference17 articles.
1. Transcriptional differences of the human papillomavirus type 16 genome between precancerous lesions and invasive carcinoma;Shinasawa;J. Virol.,1988
2. Uniformity of the splicing pattern of the E6/E7 transcripts in human papillomavirus type 16 transformed human fibroblasts, human cervical pre-malignant lesions and carcinoma;Cornelissen;J. General Virol.,1990
3. Analysis of human papillomavirus type 16 E6-E7 transcription in cervical carcinomas and normal cervical epithelium using the polymerase chain reaction;Johnson;J. General Virol.,1990
4. Human papillomavirus type 16 transformation of primary human fibroblasts requires expression of open reading frames E6 and E7;Watanabe;J. Virol.,1989
5. The E6 and E7 genes of the human papillomavirus type 16 together are necessary and sufficient for transformation of primary human keratinocytes;Munger;J. Virol.,1989
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