Senescence marker protein 30 deficiency increases Parkinson's pathology by impairing astrocyte activation-Reference-Cited by-同舟云学术

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Senescence marker protein 30 deficiency increases Parkinson's pathology by impairing astrocyte activation

Published:2013-04 Issue:4 Volume:34 Page:1177-1183
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Kim Hyun Soo,Son Tae Gen,Park Hee Ra,Lee Yonghyun,Jung Yunjin,Ishigami Akihito,Lee Jaewon

Funder

National Research Foundation of Korea

Ministry of Education, Science, and Technology

Korea government (MEST)

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference38 articles.

1. Loss of enteric dopaminergic neurons and associated changes in colon motility in an MPTP mouse model of Parkinson's disease;Anderson;Exp. Neurol.,2007

2. The kinetics and morphological characteristics of the macrophage-microglial response to kainic acid-induced neuronal degeneration;Andersson;Neuroscience,1991

3. Inactivation of tyrosine hydroxylase by nitration following exposure to peroxynitrite and 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP);Ara;Proc. Natl. Acad. Sci. U. S. A.,1998

4. Reactive astrocytes as potential manipulation targets in novel cell replacement therapy of Parkinson's disease;Chen;Curr. Drug Targets,2005

5. Senescence marker protein-30 (SMP30) rescues cell death by enhancing plasma membrane Ca(2+)-pumping activity in Hep G2 cells;Fujita;Biochem. Biophys. Res. Commun.,1998

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