Calpain and JNK pathways participate in isoflurane – induced nucleus translocation of apoptosis-inducing factor in the brain of neonatal rats-Reference-Cited by-同舟云学术

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Calpain and JNK pathways participate in isoflurane – induced nucleus translocation of apoptosis-inducing factor in the brain of neonatal rats

Published:2018-03 Issue: Volume:285 Page:60-73
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Han Xue,Liu Chuiliang,Zhang Kun,Guo Mingyan,Shen Zhiwen,Liu Yafang,Zuo Zhiyi,Cao Minghui,Li Yujuan

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Science and Technology Planning Project of Guangdong Province, China

Science and Technology Planning Project of Guangzhou, China

Medical Science and Technology Program of Foshan, China

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference74 articles.

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2. Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria;Antonsson;Biochem. J,2000

3. Calpain 10: a mitochondrial calpain and its role in calcium-induced mitochondrial dysfunction;Arrington;Am. J. Physiol. Cell Physiol.,2006

4. Mitochondrial 2', 3'-cyclic nucleotide 3'-phosphodiesterase (CNP) interacts with mPTP modulators and functional complexes (I-V) coupled with release of apoptotic factors;Baburina;Neurochem. Int.,2015

5. Isoflurane-induced neuroapoptosis in the neonatal rhesus macaque brain;Brambrink;Anesthesiology,2010

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