Inhaled ultrafine particulate matter affects CNS inflammatory processes and may act via MAP kinase signaling pathways-Reference-Cited by-同舟云学术

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Inhaled ultrafine particulate matter affects CNS inflammatory processes and may act via MAP kinase signaling pathways

Published:2008-05 Issue:2 Volume:178 Page:127-130
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Kleinman M.T.,Araujo J.A.,Nel A.,Sioutas C.,Campbell A.,Cong P.Q.,Li H.,Bondy S.C.

Funder

National Institute of Environmental Health Sciences

U.S. Environmental Protection Agency

National Institutes of Health

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference29 articles.

1. Ambient particulate pollutants in the ultrafine range promote early atherosclerosis and systemic oxidative stress;Araujo;Circulation,2008

2. Nicotine induces oxidative stress and activates nuclear transcription factor kappa B in rat mesencephalic cells;Barr;Mol. Cell Biochem.,2007

3. An inhalation exposure chamber designed for animal handling;Brown;Lab. Anim. Sci.,1981

4. Particulate matter in polluted air may increase biomarkers of inflammation in mouse brain;Campbell;Neurotoxicology,2005

5. A mouse model for human atherosclerosis: long-term histopathological study of lesion development in the aortic arch of apolipoprotein E-deficient (E0) mice;Coleman;Acta Histochem.,2006

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