Cytochrome P450-derived versus mitochondrial oxidant stress in acetaminophen hepatotoxicity-Reference-Cited by-同舟云学术

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Cytochrome P450-derived versus mitochondrial oxidant stress in acetaminophen hepatotoxicity

Published:2015-06 Issue:3 Volume:235 Page:216-217
ISSN:0378-4274
Container-title:Toxicology Letters
language:en
Short-container-title:Toxicology Letters

Author:

Jaeschke Hartmut,McGill Mitchell R.

Funder

National Institutes of Health

National Center for Research Resources

National Institute of General Medical Sciences

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference19 articles.

1. Plasma glutathione and glutathione disulfide in the rat: regulation and response to oxidative stress;Adams;J. Pharmacol. Exp. Ther.,1983

2. Acetaminophen-induced hepatotoxicity in mice occurs with inhibition of activity and nitration of mitochondrial manganese superoxide dismutase;Agarwal;J. Pharmacol. Exp. Ther.,2011

3. Expression of cytochromes P450, conjugating enzymes and nuclear receptors in human hepatoma HepaRG cells;Aninat;Drug Metab. Dispos.,2005

4. Acetaminophen-induced oxidant stress and cell injury in cultured mouse hepatocytes: protection by N-acetyl cysteine;Bajt;Toxicol. Sci.,2004

5. Peroxynitrite-induced mitochondrial and endonuclease-mediated nuclear DNA damage in acetaminophen hepatotoxicity;Cover;J. Pharmacol. Exp. Ther.,2005

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