The manipulation of cell signaling and host cell biology by cholera toxin-Reference-Cited by-同舟云学术

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The manipulation of cell signaling and host cell biology by cholera toxin

Published:2022-12 Issue: Volume:100 Page:110489
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

White Christopher,Bader Carly,Teter Ken

Funder

National Institute of Allergy and Infectious Diseases Division of Microbiology and Infectious Diseases

National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology

Reference187 articles.

1. Mutations of GS alpha designed to alter the reactivity of the protein with bacterial toxins. Substitutions at ARG187 result in loss of GTPase activity;Freissmuth;J. Biol. Chem.,1989

2. Retrograde transport of protein toxins under conditions of COPI dysfunction;Chen;Biochim. Biophys. Acta,2002

3. KDEL receptor (Erd2p)-mediated retrograde transport of the cholera toxin A subunit from the Golgi involves COPI, p23, and the COOH terminus of Erd2p;Majoul;J. Cell Biol.,1998

4. Inhibitors of COP-mediated transport and cholera toxin action inhibit simian virus 40 infection;Richards;Mol. Biol. Cell,2002

5. HSC70 and HSP90 chaperones perform complementary roles in translocation of the cholera toxin A1 subunit from the endoplasmic reticulum to the cytosol;Burress;J. Biol. Chem.,2019

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Overview of Bacterial Protein Toxins from Pathogenic Bacteria: Mode of Action and Insights into Evolution;Toxins;2024-04-08

2. Reduction is sufficient for the disassembly of ricin and Shiga toxin 1 but not Escherichia coli heat-labile enterotoxin;Infection and Immunity;2023-11-16

3. Fucosylated glycoproteins and fucosylated glycolipids play opposing roles in cholera intoxication;2023-08-03

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