Disrupting of E79 and K138 interaction is responsible for human muscle creatine kinase deficiency diseases-Reference-Cited by-同舟云学术

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Disrupting of E79 and K138 interaction is responsible for human muscle creatine kinase deficiency diseases

Published:2013-03 Issue: Volume:54 Page:216-224
ISSN:0141-8130
Container-title:International Journal of Biological Macromolecules
language:en
Short-container-title:International Journal of Biological Macromolecules

Author:

Wu Qing-Yun,Li Feng,Guo Hua-Yan,Cao Jiang,Chen Chong,Chen Wei,Zeng Ling-Yu,Li Zhen-Yu,Wang Xiao-Yun,Xu Kai-Lin

Funder

Natural Science Foundation of China

Natural Science Foundation of Jiangsu Province

China Postdoctoral Science Foundation

Publisher

Elsevier BV

Subject

Molecular Biology,General Medicine,Biochemistry,Structural Biology

Reference36 articles.

1. Creatine Kinase (Adenosine 5′-Triphosphate Creatine Phosphotrans-ferase);Watts,1973

2. Bioenergetics: Dissecting the role of creatine kinase

3. Creatine Kinase Gene Mutation in a Patient with Muscle Creatine Kinase Deficiency

4. Effects of the single point genetic mutation D54G on muscle creatine kinase activity, structure and stability

5. Amyloid β-peptide (1-42)-induced Oxidative Stress and Neurotoxicity: Implications for Neurodegeneration in Alzheimer's Disease Brain. A Review

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Roles of amino acid residues H66 and D326 in the creatine kinase activity and structural stability;International Journal of Biological Macromolecules;2018-02

2. Creatine biosynthesis and transport in health and disease;Biochimie;2015-12

3. CKM and LILRB5 Are Associated With Serum Levels of Creatine Kinase;Circulation: Cardiovascular Genetics;2014-12

4. Systems Level Regulation of Cardiac Energy Fluxes Via Metabolic Cycles: Role of Creatine, Phosphotransfer Pathways, and AMPK Signaling;Systems Biology of Metabolic and Signaling Networks;2013-08-05

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