Modification of the rate of aging of diisopropylfluorophosphate-inhibited neuropathy target esterase of hen brain
Author:
Publisher
Elsevier BV
Subject
Toxicology,General Medicine
Reference23 articles.
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2. Aldridge, W.N., 1995. Aspects of selectivity and mechanisms in neurotoxicity. In: De Matteis, F., Smith, L.L. (Eds.), Molecular and Cellular Mechanisms in Toxicity. CRC Press, Boca Raton, FL, pp. 3-17.
3. Bismith, C., Inns, R.H., Marrs, T., 1992. Efficacy, toxicity and clinical use of oximes in anticholinesterase poisoning. In: Ballantyne, B., Marrs, T.C. (Eds.), Clinical and Experimental Toxicology of Organophosphates and Carbamates. Butterworth-Heinemann, Oxford, pp. 555–577.
4. Rapid aging of neurotoxic esterase after inhibition by diisopropyl phosphorofluoridate;Clothier;Biochem. J.,1979
5. The primary biochemical lesion leading to the delayed neurotoxic effects of some organophosphorus esters;Johnson;J. Neurochem.,1974
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2. Constructs of human neuropathy target esterase catalytic domain containing mutations related to motor neuron disease have altered enzymatic properties;Toxicology Letters;2010-07-01
3. Pyridinium Oximes as Cholinesterase Reactivators in the Treatment of OP Poisoning;Handbook of Toxicology of Chemical Warfare Agents;2009
4. The Mipafox-Inhibited Catalytic Domain of Human Neuropathy Target Esterase Ages by Reversible Proton Loss;Biochemistry;2004-03-01
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