Inactivation of the lateral orbitofrontal cortex increases drinking in ethanol-dependent but not non-dependent mice-Reference-Cited by-同舟云学术

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Inactivation of the lateral orbitofrontal cortex increases drinking in ethanol-dependent but not non-dependent mice

Published:2016-08 Issue: Volume:107 Page:451-459
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

den Hartog Carolina,Zamudio-Bulcock Paula,Nimitvilai Sudarat,Gilstrap Meghin,Eaton Bethany,Fedarovich Hleb,Motts Andrew,Woodward John J.

Funder

NIH

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference30 articles.

1. Effects of chronic intermittent ethanol exposure on orbitofrontal and medial prefrontal cortex-dependent behaviors in mice;Badanich;Behav. Neurosci.,2011

2. Ethanol reduces neuronal excitability of lateral orbitofrontal cortex neurons via a glycine receptor dependent mechanism;Badanich;Neuropsychopharmacology,2013

3. Double dissociation of the effects of medial and orbital prefrontal cortical lesions on attentional and affective shifts in mice;Bissonette;J. Neurosci.,2008

4. Dorsal lesions of the prefrontal cortex: effects on alcohol consumption and subcortical monoaminergic systems;Deckel;Brain Res.,1996

5. Chronic alcohol produces neuroadaptations to prime dorsal striatal learning;DePoy;Proc. Natl. Acad. Sci. U. S. A.,2013

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