Family association study between tumour necrosis factor a gene polymorphisms and polycystic ovary syndrome in Han Chinese-Reference-Cited by-同舟云学术

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Family association study between tumour necrosis factor a gene polymorphisms and polycystic ovary syndrome in Han Chinese

Published:2014-11 Issue:5 Volume:29 Page:581-587
ISSN:1472-6483
Container-title:Reproductive BioMedicine Online
language:en
Short-container-title:Reproductive BioMedicine Online

Author:

Diao Xinghua,Han Ting,Zhang Yingchun,Ma Jinlong,Shi Yuhua,Chen Zi-Jiang

Funder

National Basic Research Program of China

National Key Technology R&D Program in the 12th Five Year Plan of China

Publisher

Elsevier BV

Subject

Developmental Biology,Reproductive Medicine,Obstetrics and Gynaecology

Reference22 articles.

1. Haploview: analysis and visualization of LD and haplotype maps;Barrett;Bioinformatics,2005

2. Polycystic ovary syndrome: current status and future perspective;Barthelmess;Front. Biosci. (Elite Ed.),2014

3. Localization and characterization of white blood cell populations within the human ovary throughout the menstrual cycle and menopause;Best;Hum. Reprod,1996

4. The role of epistasis in the etiology of polycystic ovary syndrome among Indian women: SNP-SNP and SNP-environment interactions;Dasgupta;Ann. Hum. Genet,2013

5. TNF-α haplotype association with polycystic ovary syndrome – a South Indian study;Deepika;J. Assist. Reprod. Genet,2013

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1. A kaleidoscopic view of ovarian genes associated with polycystic ovary syndrome;F&S Reviews;2021-10

2. Association of IL-1β, IL-6, TNF-α, and TGFβ1 Gene Polymorphisms with Recurrent Spontaneous Abortion in Polycystic Ovary Syndrome;Disease Markers;2020-05-05

3. Interplay between Oxidative Stress and Chronic Inflammation in PCOS: The Role of Genetic Variability in PCOS Risk and Treatment Responses;Polycystic Ovarian Syndrome;2020-04-15

4. Pathophysiologic mechanisms of obesity- and chronic inflammation-related genes in etiology of polycystic ovary syndrome;IRAN J BASIC MED SCI;2019

5. Pathogenicity of the homoplasmic C3275T, T4363C and A8343G variant requires confirmation;Gene;2019-01

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