TRPA1, but not TRPV1, is involved in the increase of the non-adrenergic non-cholinergic outflow induced by hydrogen sulfide in pithed rats

Author:

Huerta de la Cruz Saúl,Medina-Terol Grecia J.,Sánchez-López Araceli,Centurión David

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrinology,Physiology,Biochemistry

Reference43 articles.

1. The possible role of hydrogen sulfide as an endogenous neuromodulator;Abe;J. Neurosci.,1996

2. Regulation of vascular nitric oxide in vitro and in vivo; a new role for endogenous hydrogen sulphide?;Ali;Br. J. Pharm.,2006

3. Olcegepant blocks neurogenic and non-neurogenic CGRPergic vasodepressor responses and facilitates noradrenergic vasopressor responses in pithed rats;Avilés-Rosas;Br. J. Pharmacol.,2017

4. Centurión, D., Cobos-Puc, L.E., Ramírez-Rosas, M.B., Gómez-Díaz, B., Sánchez-López, A., Villalón, C.M., 2009. Pithed rat model for searching vasoactive drugs and studying the modulation of the sympathetic and non- adrenergic non-cholinergic outflow, in: Rocha, L., Granados-Soto, V. (Eds.), Models in Neuropharmacology. Kerala, India: Transworld Research Network., pp. 91–97.

5. NaHS prejunctionally inhibits the cardioaccelerator sympathetic outflow in pithed rats;Centurión;Eur. J. Pharm.,2018

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