Differential effects of estrogen exposure on arylsulfatase B, galactose-6-sulfatase, and steroid sulfatase in rat prostate development
Author:
Funder
JKT
Publisher
Elsevier BV
Subject
Cell Biology,Clinical Biochemistry,Endocrinology,Molecular Biology,Molecular Medicine,Biochemistry,Endocrinology, Diabetes and Metabolism
Reference38 articles.
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2. Arylsulfatase B (N-acetylgalactosamine-4-sulfatase): potential role as a biomarker in prostate cancer;Feferman;Prostate Cancer Prostatic Dis.,2013
3. Galectin-3 AP-1 mediate transcriptional effect of arylsulfatase B (N-acetylgalactosamine-4-sulfatase) on versican in prostate cancer;Bhattacharyya;Oncogene,2013
4. Extra-lysosomal localization of arylsulfatase B in human colonic epithelium;Prabhu;J. Histochem. Cytochem.,2011
5. Distinct effects of N-acetylgalactosamine-4-sulfatase and galactose-6-sulfatase expression on chondroitin sulfate;Bhattacharyya;J. Biol. Chem.,2008
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1. Profound Impact of Decline in N-Acetylgalactosamine-4-Sulfatase (Arylsulfatase B) on Molecular Pathophysiology and Human Diseases;International Journal of Molecular Sciences;2022-10-29
2. Molecular environment and atypical function: What do we know about enzymes associated with Mucopolysaccharidoses?;Orphanet Journal of Rare Diseases;2022-03-04
3. Dihydrotestosterone inhibits arylsulfatase B and Dickkopf Wnt signaling pathway inhibitor (DKK)‐3 leading to enhanced Wnt signaling in prostate epithelium in response to stromal Wnt3A;The Prostate;2019-02-22
4. Chondroitin sulfatases differentially regulate Wnt signaling in prostate stem cells through effects on SHP2, phospho-ERK1/2, and Dickkopf Wnt signaling pathway inhibitor (DKK3);Oncotarget;2017-10-27
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