NLRP3 inflammasome activation triggers severe inflammatory liver injury in N, N-dimethylformamide-exposed mice
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Reference77 articles.
1. Redox regulation of NLRP3 inflammasomes: ROS as trigger or effector?;Abais;Antioxid. Redox Signal.,2015
2. Cutting edge: reactive oxygen species inhibitors block priming, but not activation, of the NLRP3 inflammasome;Bauernfeind;J. Immunol.,2011
3. NLRP3 deletion inhibits the non-alcoholic steatohepatitis development and inflammation in Kupffer cells induced by palmitic acid;Cai;Inflammation,2017
4. Study of N-methylcarbamoylated hemoglobin in blood of being an exposure biomarker of N, N-dimethylformamide;Chen;Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi,2014
5. Reactive oxygen species and mitochondria: a nexus of cellular homeostasis;Dan Dunn;Redox Biol.,2015
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