Genome-wide association studies suggest that APOL1-environment interactions more likely trigger kidney disease in African Americans with nondiabetic nephropathy than strong APOL1–second gene interactions-Reference-Cited by-同舟云学术

Genome-wide association studies suggest that APOL1-environment interactions more likely trigger kidney disease in African Americans with nondiabetic nephropathy than strong APOL1–second gene interactions

Published:2018-09 Issue:3 Volume:94 Page:599-607
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Langefeld Carl D.,Comeau Mary E.,Ng Maggie C.Y.,Guan Meijian,Dimitrov Latchezar,Mudgal Poorva,Spainhour Mitzie H.,Julian Bruce A.,Edberg Jeffrey C.,Croker Jennifer A.,Divers Jasmin,Hicks Pamela J.,Bowden Donald W.,Chan Gary C.,Ma Lijun,Palmer Nicholette D.,Kimberly Robert P.,Freedman Barry I.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Nephrology

Reference36 articles.

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2. Gene-gene and gene-environment interactions in apolipoprotein L1 gene-associated nephropathy;Freedman;Clin J Am Soc Nephrol,2014

3. Therapeutics for APOL1 nephropathies: putting out the fire in the podocyte;Heymann;Nephrol Dial Transplant,2017

4. APOL1 risk variants are strongly associated with HIV-associated nephropathy in black South Africans;Kasembeli;J Am Soc Nephrol,2015

5. APOL1 genetic variants in focal segmental glomerulosclerosis and HIV-associated nephropathy;Kopp;J Am Soc Nephrol,2011

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