Increased soluble fms-like tyrosine kinase 1 after ischemia reperfusion contributes to adverse clinical outcomes following kidney transplantation-Reference-Cited by-同舟云学术

Increased soluble fms-like tyrosine kinase 1 after ischemia reperfusion contributes to adverse clinical outcomes following kidney transplantation

Published:2019-05 Issue:5 Volume:95 Page:1091-1102
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Wewers Theresa M.,Mayer Anna B.,Pfleiderer Alexander,Beul Katrin,Schmidt Rene,Heitplatz Barbara,Van Marck Veerle,Nolte Ingo,Pavenstädt Hermann,Reuter Stefan,Brand Marcus,Di Marco Giovana S.^ORCID

Funder

Innovative Medizinische Forschung

Publisher

Elsevier BV

Subject

Nephrology

Reference50 articles.

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2. Cellular pathophysiology of ischemic acute kidney injury;Bonventre;J Clin Invest,2011

3. The endothelial cell in ischemic acute kidney injury: implications for acute and chronic function;Basile;Kidney Int,2007

4. Acute renal failure;Lameire;Lancet,2005

5. Difficulties in understanding human “acute tubular necrosis”: limited data and flawed animal models;Rosen;Kidney Int,2001

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1. Role of extracellular vesicles in pathogenesis and therapy of renal ischemia-reperfusion injury;Biomedicine & Pharmacotherapy;2023-09

2. The Endothelial Glycocalyx as a Target of Excess Soluble Fms-like Tyrosine Kinase-1;International Journal of Molecular Sciences;2023-03-11

3. Angiogenesis under Opioids Preconditioning in Renal Ischemia Reperfusion;Drugs and Drug Candidates;2023-01-07

4. The Soluble Fms-like Tyrosine Kinase-1 Contributes to Structural and Functional Changes in Endothelial Cells in Chronic Kidney Disease;International Journal of Molecular Sciences;2022-12-16

5. sFlt-1 in Chronic Kidney Disease: Friend or Foe?;International Journal of Molecular Sciences;2022-11-16