Differential effects of triethyllead on synaptosomal [3H]dopamine vs. [3H]acetylcholine and [3H]γ-aminobutyric acid release

Author:

Minnema Daniel J.,Cooper Gary P.,Schamer Marlene M.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Developmental Neuroscience,Toxicology

Reference34 articles.

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4. A note on the mechanisms by which inhibitors of the sodium pump accelerate spontaneous release of transmitter from motor nerve terminals;Baker;J. Physiol. (Lond.),1975

5. The neurotoxicity of organic and inorganic lead;Bondy,1988

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1. Safety and Environmental Effects;PATAI'S Chemistry of Functional Groups;2009-12-15

2. The importance of glutamate, glycine, and γ-aminobutyric acid transport and regulation in manganese, mercury and lead neurotoxicity;Toxicology and Applied Pharmacology;2005-05

3. Triethyllead decreases central benzodiazepine receptor binding in rat cerebellum ex vivo;European Journal of Pharmacology: Environmental Toxicology and Pharmacology;1995-07

4. Neurotoxic Metals and Neuronal Signalling Processes;The Vulnerable Brain and Environmental Risks;1992

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