Chimerization at the AQP2–AQP3 locus is the genetic basis of melarsoprol–pentamidine cross-resistance in clinical Trypanosoma brucei gambiense isolates-Reference-Cited by-同舟云学术

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Chimerization at the AQP2–AQP3 locus is the genetic basis of melarsoprol–pentamidine cross-resistance in clinical Trypanosoma brucei gambiense isolates

Published:2015-08 Issue:2 Volume:5 Page:65-68
ISSN:2211-3207
Container-title:International Journal for Parasitology: Drugs and Drug Resistance
language:en
Short-container-title:International Journal for Parasitology: Drugs and Drug Resistance

Author:

Graf Fabrice E.,Baker Nicola,Munday Jane C.,de Koning Harry P.,Horn David^ORCID,Mäser Pascal

Funder

National Science Foundation

Medical Research Council

Department for International Development, UK

Wellcome Trust

Publisher

Elsevier BV

Subject

Pharmacology (medical),Infectious Diseases,Pharmacology,Parasitology

Reference21 articles.

1. Tagging a T. brucei RRNA locus improves stable transfection efficiency and circumvents inducible expression position effects;Alsford;Mol. Biochem. Parasitol,2005

2. High-throughput phenotyping using parallel sequencing of RNA interference targets in the African trypanosome;Alsford;Genome Res,2011

3. High-throughput decoding of antitrypanosomal drug efficacy and resistance;Alsford;Nature,2012

4. Aquaglyceroporin 2 controls susceptibility to melarsoprol and pentamidine in African trypanosomes;Baker;Proc. Natl. Acad. Sci. U.S.A.,2012

5. Drug resistance in African trypanosomiasis: the melarsoprol and pentamidine story;Baker;Trends Parasitol,2013

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