Melatonin improves nitric oxide bioavailability in isoproterenol induced myocardial injury

Author:

Santos Ramison,Turck Patrick,de Mello Palma VictorORCID,Visioli FernandaORCID,Ortiz Vanessa Duarte,Proença Isabel Cristina Teixeira,Fernandes Tânia Regina G.,Fernandes Elissa,Tasca Silvio,Carraro Cristina Campos,Belló-Klein Adriane,da Rosa Araujo Alex Sander,Khaper Neelam,de Castro Alexandre Luz

Funder

Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Universidade Federal do Rio Grande do Sul

Publisher

Elsevier BV

Reference47 articles.

1. Simvastatin prevents isoproterenol-induced cardiac hypertrophy through modulation of the JAK/STAT pathway;Al-Rasheed;Drug Des. Dev. Ther.,2015

2. Umbelliferone prevents isoproterenol-induced myocardial injury by upregulating Nrf2/HO-1 signaling, and attenuating oxidative stress, inflammation, and cell death in rats;Althunibat;Biomed. Pharmacother.,2022

3. Cardioprotective melatonin: translating from proof-of-concept studies to therapeutic use;Baltatu;Int. J. Mol. Sci.,2019

4. Melatonin, mitochondria and hypertension;Baltatu;Cell. Mol. Life Sci.,2017

5. Melatonin influences NO/NOS pathway and reduces oxidative and nitrosative stress in a model of hypoxic-ischemic brain damage;Blanco;Nitric Oxide,2017

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