p19INK4d inhibits proliferation and enhances imatinib efficacy through BCR-ABL signaling pathway in chronic myeloid leukemia-Reference-Cited by-同舟云学术

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p19INK4d inhibits proliferation and enhances imatinib efficacy through BCR-ABL signaling pathway in chronic myeloid leukemia

Published:2020-11 Issue: Volume:85 Page:102477
ISSN:1079-9796
Container-title:Blood Cells, Molecules, and Diseases
language:en
Short-container-title:Blood Cells, Molecules, and Diseases

Author:

Kuang Yijin,Han Xu,Cao Pengfei,Xiong Dehui,Peng Yuanliang,Liu Zhaoping,Xu Zhenru,Liang Long,Roy Mridul,Liu Jing,Nie Ling,Zhang Ji

Funder

National Natural Science Foundation of China

Hunan Provincial Health Commission

National Key Research and Development Program of China

Central South University

Publisher

Elsevier BV

Subject

Cell Biology,Hematology,Molecular Biology,Molecular Medicine

Reference50 articles.

1. Letter: a new consistent chromosomal abnormality in chronic myelogenous leukaemia identified by quinacrine fluorescence and Giemsa staining;Rowley;Nature,1973

2. Chronic myeloid leukaemia;Apperley;Lancet (London, England),2015

3. A cellular oncogene is translocated to the Philadelphia chromosome in chronic myelocytic leukaemia;de Klein;Nature,1982

4. Arsenic sulfide nanoformulation induces erythroid differentiation in chronic myeloid leukemia cells through degradation of BCR-ABL;Wang;Int. J. Nanomedicine,2019

5. Nickel pyrithione induces apoptosis in chronic myeloid leukemia cells resistant to imatinib via both Bcr/Abl-dependent and Bcr/Abl-independent mechanisms;Lan;J. Hematol. Oncol.,2016

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