The closely related small nuclear ribonucleoprotein polypeptides N and B/B' are distinguishable by antibodies as well as by differences in their mRNAs and gene structures.-Reference-Cited by-同舟云学术

The closely related small nuclear ribonucleoprotein polypeptides N and B/B' are distinguishable by antibodies as well as by differences in their mRNAs and gene structures.

Published:1990-06 Issue:18 Volume:265 Page:10733-10739
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Schmauss C,Lerner M R

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference34 articles.

1. Structure and Function of Major and Minor Small Nuclear Ribonuclear Ribonucleoprotein Particles;Steitz,1988

2. Purification of the individual snRNPs U1, U2, U5 and U4/U6 from HeLa cells and characterization of their protein constituents.

3. Antibodies to small nuclear RNAs complexed with proteins are produced by patients with systemic lupus erythematosus

4. Autoantibodies to Nuclear Antigens (ANA): Their Immunobiology and Medicine

5. Molecular relationships between U snRNP proteins as investigated by rabbit antisera and peptide mapping

Cited by 20 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Antibodies to specific EBNA-1 domains and HLA DRB1⁎1501 interact as risk factors for multiple sclerosis;Journal of Neuroimmunology;2009-10

2. The symmetrical dimethylarginine post-translational modification of the SmD3 protein is not required for snRNP assembly and nuclear transport;Biochemical and Biophysical Research Communications;2005-12

3. LSm proteins form heptameric rings that bind to RNA via repeating motifs;Trends in Biochemical Sciences;2005-09

4. Mouse Tudor Repeat-1 (MTR-1) is a novel component of chromatoid bodies/nuages in male germ cells and forms a complex with snRNPs;Mechanisms of Development;2003-09

5. Paternal Deletion from Snrpn to Ube3a in the Mouse Causes Hypotonia, Growth Retardation and Partial Lethality and Provides Evidence for a Gene Contributing to Prader-Willi Syndrome;Human Molecular Genetics;1999-08-01