The role of apoptosis in acetaminophen hepatotoxicity
Author:
Funder
National Institute of General Medical Sciences
National Institutes of Health
Publisher
Elsevier BV
Subject
Toxicology,General Medicine,Food Science
Reference133 articles.
1. Enhanced acetaminophen hepatotoxicity in transgenic mice overexpressing BCL-2;Adams;Mol. Pharmacol.,2001
2. Overexpression of aldose reductase render mouse hepatocytes more sensitive to acetaminophen induced oxidative stress and cell death;Ahmed;Indian J. Clin. Biochem.,2016
3. Optimization of the HepaRG cell model for drug metabolism and toxicity studies;Anthérieu;Toxicol. Vitro,2012
4. Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity;Antoine;J. Hepatol.,2012
5. Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity;Antoine;Mol. Med. (Tokyo),2010
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