α-Synuclein misfolding and aggregation: Implications in Parkinson’s disease pathogenesis-Reference-Cited by-同舟云学术

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α-Synuclein misfolding and aggregation: Implications in Parkinson’s disease pathogenesis

Published:2019-10 Issue:10 Volume:1867 Page:890-908
ISSN:1570-9639
Container-title:Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics

Author:

Mehra Surabhi,Sahay Shruti,Maji Samir K.

Funder

Department of Biotechnology

Council of Scientific & Industrial Research

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics,Analytical Chemistry

Reference308 articles.

1. Aggregation of α-synuclein in Lewy bodies of sporadic Parkinson's disease and dementia with Lewy bodies;Baba;Am J Pathol,1998

2. α-Synuclein in Lewy bodies;Spillantini;Nature,1997

3. Overexpression of human α-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells;Zhou;Brain Res,2000

4. Dopamine-dependent neurotoxicity of α-synuclein: a mechanism for selective neurodegeneration in Parkinson disease;Xu;Nat Med,2002

5. A Drosophila model of Parkinson's disease;Feany;Nature,2000

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