Epidemiologic, molecular, and functional evidence suggest A572D-SCN5A should not be considered an independent LQT3-susceptibility mutation-Reference-Cited by-同舟云学术

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Epidemiologic, molecular, and functional evidence suggest A572D-SCN5A should not be considered an independent LQT3-susceptibility mutation

Published:2010-07 Issue:7 Volume:7 Page:912-919
ISSN:1547-5271
Container-title:Heart Rhythm
language:en
Short-container-title:Heart Rhythm

Author:

Tester David J.,Valdivia Carmen,Harris-Kerr Carole,Alders Marielle,Salisbury Benjamin A.,Wilde Arthur A.M.,Makielski Jonathan C.,Ackerman Michael J.

Publisher

Elsevier BV

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference23 articles.

1. Prevalence of the congenital long-QT syndrome;Schwartz;Circulation,2009

2. Risk stratification in the long-QT syndrome;Priori;N Engl J Med,2003

3. Modulating effects of age and gender on the clinical course of long QT syndrome by genotype;Zareba;J Am Coll Cardiol,2003

4. Therapeutic strategies for long-QT syndrome: does the molecular substrate matter?;Ruan;Circ Arrhythm Electrophysiol,2008

5. Genomic structure of three long QT syndrome genes: KVLQT1, HERG, and KCNE1;Splawski;Genomics,1998

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