SEMA3A as a biomarker of primary ventricular fibrillation complicating STEMI-Reference-Cited by-同舟云学术

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SEMA3A as a biomarker of primary ventricular fibrillation complicating STEMI

Published:2024-08 Issue: Volume: Page:
ISSN:1547-5271
Container-title:Heart Rhythm
language:en
Short-container-title:Heart Rhythm

Author:

BOITEUX Clément^ORCID,LEBOUBE Simon,HAYEK Ahmad,BRUN Camille,BESSIERE Francis,MEWTON Nathan,BIDAUX Gabriel,CROLA DA SILVA Claire,CHEVALIER Philippe,BOCHATON Thomas

Funder

Fédération Française de Cardiologie

French National Research Agency

Publisher

Elsevier BV

Reference4 articles.

1. Overexpression of sema3a in myocardial infarction border zone decreases vulnerability of ventricular tachycardia post-myocardial infarction in rats;Chen;Journal of Cellular and Molecular Medicine,2013

2. Semaphorin 3A attenuates cardiac autonomic disorders and reduces inducible ventricular arrhythmias in rats with experimental myocardial infarction;Hu;BMC Cardiovasc Disord,2016

3. Sema3A promotes the resolution of cardiac inflammation after myocardial infarction;Rienks;Basic Res Cardiol,2017

4. A Nonsynonymous Polymorphism in Semaphorin 3A as a Risk Factor for Human Unexplained Cardiac Arrest with Documented Ventricular Fibrillation;Nakano;PLOS Genetics,2013

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