Enprostil reduces the increase of gastric corpus mucosal mass induced by the hydrogen-potassium-stimulated adenosine triphosphatase inhibitor BY 831-78 in the rat-Reference-Cited by-同舟云学术

Enprostil reduces the increase of gastric corpus mucosal mass induced by the hydrogen-potassium-stimulated adenosine triphosphatase inhibitor BY 831-78 in the rat

Published:1989-10 Issue:4 Volume:97 Page:846-852
ISSN:0016-5085
Container-title:Gastroenterology
language:en
Short-container-title:Gastroenterology

Author:

Inauen W.,Rohner C.,Koelz H.R.,Herdmann J.,Schürer-Maly C-C.,Varga L.,Halter F.

Publisher

Elsevier BV

Subject

Gastroenterology,Hepatology

Reference28 articles.

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3. Dose-response study of omeprazole on meal-stimulated gastric acid secretion and gastrin release;Londong;Gastroenterology,1983

4. Effect of single and repeated doses of oral omeprazole on gastric acid and pepsin secretion and fasting serum gastrin and serum pepsinogen I levels;Festen;Dig Dis Sci,1986

5. Hypergastrinemia produces trophic effects in stomach but not in pancreas and intestines;Hakanson;Regul Pept,1986

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1. Effect of acute and chronic acid suppression on plasma gastrin release in the rat;Alimentary Pharmacology & Therapeutics;2007-03-31

2. Attenuation of the gastric acid and serum gastrin – lowering effects of the prostaglandin E2 analog enprostil;Clinical Pharmacology and Therapeutics;1993-06

3. Aspects of the Role of Prostaglandins in Gastrin Histamine Regulation of Gastric Acid Secretion;Scandinavian Journal of Gastroenterology;1991-01

4. Effect of 16.16 Dimethyl Prostaglandin E₂, N-Acetyl-Cysteine and the Proton Pump Inhibitor BY 831–78 on Hydrogen Peroxide-Induced Mucosal Damage in the Rat Stomach;Digestion;1990

5. Chapter 17. Agents for the Treatment of Peptic Ulcer Dlsease;Annual Reports in Medicinal Chemistry;1990