The Archaeal XPB Protein is a ssDNA-Dependent ATPase with a Novel Partner
Author:
Publisher
Elsevier BV
Subject
Molecular Biology,Structural Biology
Reference40 articles.
1. TFIIH enzymatic activities in transcription and nucleotide excision repair;Laine;Methods Enzymol.,2006
2. Mutations in XPB and XPD helicases found in xeroderma pigmentosum patients impair the transcription function of TFIIH;Coin;EMBO J.,1999
3. TFIIH with inactive XPD helicase functions in transcription initiation but is defective in DNA repair;Winkler;J. Biol. Chem.,2000
4. Distinct roles for the XPB/p52 and XPD/p44 subcomplexes of TFIIH in damaged DNA opening during nucleotide excision repair;Coin;Mol. Cell,2007
5. Mutations in XPD helicase prevent its interaction and regulation by p44, another subunit of TFIIH, resulting in xeroderma pigmentosum (XP) and trichothiodystrophy (TTD) phenotypes;Coin;Pathol. Biol. (Paris),1998
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