Oncostatin M Improves Cutaneous Wound Re-Epithelialization and Is Deficient under Diabetic Conditions-Reference-Cited by-同舟云学术

Oncostatin M Improves Cutaneous Wound Re-Epithelialization and Is Deficient under Diabetic Conditions

Published:2021-09 Issue: Volume: Page:
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Das Amitava^ORCID,Madeshiya Amit K.,Biswas Nirupam^ORCID,Ghosh Nandini^ORCID,Gorain Mahadeo^ORCID,Rawat Atul^ORCID,Mahajan Sanskruti P.^ORCID,Khanna Savita^ORCID,Sen Chandan K.^ORCID,Roy Sashwati^ORCID

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference55 articles.

1. Improvement of human keratinocyte migration by a redox active bioelectric dressing;Banerjee;PLoS One,2014

2. Oncostatin M mediates STAT3-dependent intestinal epithelial restitution via increased cell proliferation, decreased apoptosis and upregulation of SERPIN family members;Beigel;PLoS One,2014

3. Hypoxia inducible microRNA 210 attenuates keratinocyte proliferation and impairs closure in a murine model of ischemic wounds;Biswas;Proc Natl Acad Sci USA,2010

4. Oncostatin M secreted by skin infiltrating T lymphocytes is a potent keratinocyte activator involved in skin inflammation;Boniface;J Immunol,2007

5. Increased KGF expression promotes fibroblast activation in a double paracrine manner resulting in cutaneous fibrosis;Canady;J Invest Dermatol,2013

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