Pharmacological investigations of the cellular transduction pathways used by cholecystokinin to activate nodose neurons
Author:
Funder
NIH
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Endocrine and Autonomic Systems
Reference50 articles.
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4. 2-aminoethoxydiphenyl borate (2-APB) is a reliable blocker of store-operated Ca2+ entry but an inconsistent inhibitor of InsP3-induced Ca2+ release;Bootman;FASEB J.,2002
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1. TRPV1 enhances cholecystokinin signaling in primary vagal afferent neurons and mediates the central effects on spontaneous glutamate release in the NTS;American Journal of Physiology-Cell Physiology;2024-01-01
2. Contributing mechanisms underlying desensitization of cholecystokinin-induced activation of primary nodose ganglia neurons;American Journal of Physiology-Cell Physiology;2020-04-01
3. Persistent cytosolic Ca2+ increase induced by angiotensin II at nanomolar concentrations in acutely dissociated subfornical organ (SFO) neurons of rats;Brain Research;2019-09
4. Occipital Artery Function during the Development of 2-Kidney, 1-Clip Hypertension in Rats;International Journal of Vascular Medicine;2014
5. Phospholipase C not protein kinase C is required for the activation of TRPC5 channels by cholecystokinin;European Journal of Pharmacology;2012-08
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