Programmed cell death‐2 isoform1 is ubiquitinated by parkin and increased in the substantia nigra of patients with autosomal recessive Parkinson's disease-Reference-Cited by-同舟云学术

Programmed cell death‐2 isoform1 is ubiquitinated by parkin and increased in the substantia nigra of patients with autosomal recessive Parkinson's disease

Published:2009-01-13 Issue:3 Volume:583 Page:521-525
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:FEBS Letters

Author:

Fukae Jiro¹,Sato Shigeto¹,Shiba Kahori²,Sato Ken-ichi¹,Mori Hideo¹,Sharp Philip A³,Mizuno Yoshikuni²,Hattori Nobutaka¹

Affiliation:

1. Department of Neurology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo, Tokyo 113-8421, Japan

2. Research Institute for Diseases of Old Ages, Juntendo University School of Medicine, Tokyo, Japan

3. Center for Cancer Research, Department of Biology, Massachusetts Institute of Technology, 77 Massachusetts Avenue, Cambridge, MA 02139-4307, USA

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1016/j.febslet.2008.12.055

Reference14 articles.

1. Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

2. Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase

3. Identification of mRNAs associated with programmed cell death in immature thymocytes.

4. Isolation and mapping of a human gene (PDCD2) that is highly homologous to Rp8, a rat gene associated with programmed cell death

5. Expression of the death-associated gene RP-8 in granule cell neurons undergoing postnatal cell death in the cerebellum of weaver mice

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