Serum from pemphigus vulgaris reduces desmoglein 3 half-life and perturbs its de novo assembly to desmosomal sites in cultured keratinocytes
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
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3. Explanations for the clinical and microscopic localization of lesions in pemphigus foliaceus and vulgaris;Mahoney;J. Clin. Invest.,1999
4. Pemphigus IgG, but not bullous pemphigoid IgG, causes a transient increase in intracellular calcium and inositol 1,4,5-triphosphate in DJM-1 cells, a squamous cell carcinoma line;Seishima;J. Invest. Dermatol.,1995
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1. Autoantibody-Specific Signalling in Pemphigus;Frontiers in Medicine;2021-08-09
2. Plakophilin 1 but not plakophilin 3 regulates desmoglein clustering;Cellular and Molecular Life Sciences;2019-04-04
3. Hyper-adhesion: A Unique Property of Desmosomes;Cell Communication & Adhesion;2014-06-30
4. Desmosomal Adhesion In Vivo;Cell Communication & Adhesion;2014-01-24
5. MAPKAP Kinase 2 (MK2)-Dependent and -Independent Models of Blister Formation in Pemphigus Vulgaris;Journal of Investigative Dermatology;2014-01
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