Detection of new anti-neutral glycosphingolipids antibodies and their effects on Trk neurotrophin receptors-Reference-Cited by-同舟云学术

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Detection of new anti-neutral glycosphingolipids antibodies and their effects on Trk neurotrophin receptors

Published:2006-08-17 Issue:21 Volume:580 Page:4991-4995
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Mihara T.,Ueda A.,Hirayama M.,Takeuchi T.,Yoshida S.,Naito K.,Yamamoto H.,Mutoh T.

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/j.febslet.2006.08.013/fullpdf

Reference18 articles.

1. Peripheral neuropathies and anti-glycolipid antibodies;Willison;Brain,2002

2. Ganglioside function in the neuron;Ledeen;Trends Glycosci. Glycotech.,1992

3. Anti-Gal-C antibodies in GBS subsequent to mycoplasma infection: evidence of molecular mimicry;Kusunoki;Neurology,2001

4. Glycolipid-enriched caveolae and caveolae-like domains in the nervous system;Masserini;J. Neurochem.,1999

5. Vital role for glycosphingolipid synthesis during development and differentiation;Yamashita;Proc. Natl. Acad. Sci. USA,1999

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1. Sphingolipid abnormalities in encephalomyeloradiculoneuropathy (EMRN) are associated with an anti‐neutral glycolipid antibody;FEBS Open Bio;2023-03-02

2. Neutral Glycosphingolipids As Neuroinflammatory Signaling Molecules In Neurodegeneration;Trends in Glycoscience and Glycotechnology;2021-01-25

3. Comparison of relapsing polychondritis patients with and without central nervous system involvement: A retrospective study of 181 patients;International Journal of Immunopathology and Pharmacology;2021-01

4. GM1 Ganglioside Is A Key Factor in Maintaining the Mammalian Neuronal Functions Avoiding Neurodegeneration;International Journal of Molecular Sciences;2020-01-29

5. Severe visual impairment and subclinical encephalitis preceding clinical signs of chondritis in relapsing polychondritis;Neurology and Clinical Neuroscience;2018-11-14

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