A Proposed Mechanism for Neurodegeneration in Movement Disorders Characterized by Metal Dyshomeostasis and Oxidative Stress-Reference-Cited by-同舟云学术

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A Proposed Mechanism for Neurodegeneration in Movement Disorders Characterized by Metal Dyshomeostasis and Oxidative Stress

Published:2018-07 Issue:7 Volume:25 Page:807-816
ISSN:2451-9456
Container-title:Cell Chemical Biology
language:en
Short-container-title:Cell Chemical Biology

Author:

Trist Benjamin Guy,Hare Dominic James,Double Kay Lorraine

Funder

Parkinson’s NSW

University of Sydney

National Health and Medical Research Council

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Biology,Molecular Medicine,Biochemistry

Reference91 articles.

1. Ceruloplasmin dysfunction and therapeutic potential for Parkinson disease;Ayton;Ann. Neurol.,2013

2. SOD1 and amyotrophic lateral sclerosis: mutations and oligomerization;Banci;PLoS One,2008

3. Metal-free superoxide dismutase forms soluble oligomers under physiological conditions: a possible general mechanism for familial ALS;Banci;Proc. Natl. Acad. Sci. USA,2007

4. Oxidative stress in ALS: key role in motor neuron injury and therapeutic target;Barber;Free Radic. Biol. Med.,2010

5. Mechanisms of biosynthesis of mammalian copper/zinc superoxide dismutase;Bartnikas;J. Biol. Chem.,2003

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