Bisphenol A induces focal adhesions assembly and activation of FAK, Src and ERK2 via GPER in MDA-MB-231 breast cancer cells-Reference-Cited by-同舟云学术

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Bisphenol A induces focal adhesions assembly and activation of FAK, Src and ERK2 via GPER in MDA-MB-231 breast cancer cells

Published:2020-08 Issue: Volume:66 Page:104871
ISSN:0887-2333
Container-title:Toxicology in Vitro
language:en
Short-container-title:Toxicology in Vitro

Author:

Castillo-Sanchez Rocio,Ramirez-Ricardo Javier,Martinez-Baeza Elia,Cortes-Reynosa Pedro,Candanedo-Gonzales Fernando,Gomez Rocio,Salazar Eduardo Perez

Funder

CONACYT-FOSISS-Mexico

CONACYT-Mexico

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference47 articles.

1. Focal adhesions: a personal perspective on a half century of progress;Burridge;FEBS J.,2017

2. Tyrosine phosphorylation of focal adhesion kinase at sites in the catalytic domain regulates kinase activity: a role for Src family kinases;Calalb;Mol. Cell. Biol.,1995

3. A novel role for FAK as a protease-targeting adaptor protein: regulation by p42 ERK and Src;Carragher;Curr. Biol.,2003

4. Bisphenol A induces migration through a GPER-, FAK-, Src-, and ERK2-dependent pathway in MDA-MB-231 breast cancer cells;Castillo Sanchez;Chem. Res. Toxicol.,2016

5. Activation of Src in human breast tumor cell lines: elevated levels of phosphotyrosine phosphatase activity that preferentially recognizes the Src carboxy terminal negative regulatory tyrosine 530;Egan;Oncogene,1999

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3. Comprehensive understanding of the role of GPER in estrogen receptor-alpha negative breast cancer;The Journal of Steroid Biochemistry and Molecular Biology;2024-07

4. Bisphenols and Their Interaction with GPER-1: The Invisible Enemy Behind Breast Cancer and Its Societal Impact;Pharmaceutical Science;2024-03-11

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